Showing posts with label Mnemonics. Show all posts
Showing posts with label Mnemonics. Show all posts

September 23, 2013

FUN Teaching Principles (based on learning theories)

Teaching is the process in which learning is facilitated through planning, presentation, observation, active reflection and feedback that results in external stimuli being perceived, translated, converted and comprehended in a manner which results in cognitive and/or behavioural changes.

Kaufman and Mann’s ‘Teaching and Learning in Medical Education’ chapter in “Understanding Medical Education” inspires this personal definition of teaching.  My interpretation reflects a cognitive constructivist philosophy in which the teacher and learner engage in a “continuous, dynamic, reciprocal interaction among three sets of determinants: personal, environmental (situational) and behavioural.” (Kaufman & Mann 2010)  The personal factors are the behind-the-scenes models/schemas the learner mentally constructs, the environmental factors are the learning setting/material and the behavioural factors primarily are the outcomes, building on prior knowledge.

My philosophy of teaching guided me to derive a set of simple teaching principles that form a simple mnemonic: FUN!
·         First things First (Planning and Presentation)
·         Understand the Learner (Observation)
·         Nurture and Guide (Active Reflection and Feedback)
These principles are not a step-by-step guide to develop a lesson plan, but they provide a framework for considering elements of cognitivist and social constructivist perspectives.  I will elucidate these points and provide an example from a lesson plan I developed with second-year residents on office efficiency.

First Things First:
Before a teacher picks up a set of learning objectives/syllabus or launches Powerpoint to make lecture notes, it is important to reflect and set priorities.  A series of questions based on Schön’s Reflective Practice (1983) may help create a learner-centered lesson plan.

Question
Schön’s Reflective Practice
What was MY EXPERIENCE when I first started this subject?
Knowing-in-action
What did I find most CHALLENGING initially?
Surprise
What did I learn that was most IMPORTANT to me at that time?
Reflection-in-action
How has my understanding of this subject CHANGED since then?
Experimentation
How do I use the subject-matter on a DAY-TO-DAY basis?
Reflection-on-action

These questions walk a teacher through a mental progression starting as a novice learner, synthesizing the content into a mental model and then applying it.  Schön’s steps can help a teacher to recall a time when he or she was an early learner so as to avoid the expert’s pitfall of unconscious competence: taking mental short cuts and making assumptions that novices find difficult.
For example, when I created a lesson plan to teach second year residents how to function efficiently in the outpatient clinic, I first stepped back and recalled how I felt at that time in my learning.  I was overwhelmed and often fell behind because of the increased load of patients that I had to see in 20-minute segments.  I often felt I survived the day only by finishing hasty notes that felt sparse and inadequate.  I learned tools and workflows from co-residents and a teaching fellow.  Eventually with time and reflection, I was able to change my practices to shift from a 40-minute visit per patient to a more efficient 20-minute visit per patient mentality.
Based on this reflection, I decided that my lesson planning would follow a “typical day in clinic”: I would have each resident read and react to scenarios that progress through a normal resident’s workday.  The teaching would focus on practical issues like chart review/prep-work, lecture note-taking/review, and clinic/charting workflows, while reflecting on the frustrations of time management, chart closing, and difficult patients.


Understand the Learner:
While the first step emphasizes the teacher’s personal perspective to assist with priority setting and lesson planning, Understanding the Learner shifts the focus to learners’ perspectives to understand what learning styles may need to be considered in the lesson.  The Kolb Learning Cycle (1984) and the Honey and Mumford adaptation close the gaps through an experiential learning approach, building in elements that reinforce the lessons to be learned long-term.  During the process of lesson planning, the teacher considers where students enter the learning cycle and engages them using a variety of approaches.


Accessed 9/2013 from: http://bonlinelearning.com.au/blog/learning-styles-in-elearning/

For example, in my lesson on office efficiency, I devised various case scenarios from a typical day to access Kolb’s “concrete experience” and “reflective observation” stages.  The group’s discussions accessed “abstract conceptualization” and their homework and subsequent ‘real work’ would access “active experimentation” to complete Kolb’s learning cycle.  These steps mirrored the reflective practice model as well: as each student read their scenario out loud, they reflected on what they would do (virtual reflection-in-action), discussed strategies with other R2s (experimentation) and then as homework, they created/utilized/tweaked personal workflows to gauge if there were any improvements (reflection-on-action).


Nurture and Guide:
Using the ‘clinic as the curriculum’ is a prime driver for my residency and this orientation helps facilitate individual learners to become self-directed adult learners and information masters, drawing strongly from an Adult Learning Theory/Andragogy model.  Knowles’ principles (1984) encourage a fun and safe environment, resident engagement in diagnosing learning needs and developing their own learning resources, and aiding them in carrying out their learning plan.  A curriculum that promotes an open, accepting, transparent and sharing culture of learning promotes a strong Community of Practice. (Wenger 1998)  Scaffolding occurs by building on prior knowledge and utilizing higher learners in a Zone of Proximal Development (Vygotsky 1986).


Accessed 9/2013: http://lmrtriads.wikispaces.com/Zone+of+Proximal+Development

Initially, the suggestion to learn more about office efficiency came from the residents as they were transitioning to more solid roles within the clinic.  This is an example of andragogy, wherein the R2s actively engaged with their curriculum development.  I deliberately crafted the office efficiency lesson so they would scaffold each other’s learning using principles of social constructivism.
The scenarios I created provide a framework: embedded within the cases are best practices that they share aloud.  The scenarios end with a point of contention: questions like “what do you do to keep yourself organized and how do you stay on track [with clinic flow]?” and “what sort of preparation work do you do tonight? The morning before [you see your patients]?”  If they are stumped, they can turn to me as a resource: as a fellow, I am a step between residency and attending, placing me in the role of a “more knowledgeable other.”  My recent experiences make me receptive to their needs and subsequently, they are receptive to my pearls.

In conclusion, the teaching principles “First things First,” “Understand the Learner” and “Nurture & Guide” or FUN, form the basis for my teaching philosophy.  Their application reflects the teaching theories of andragogy, reflective practice, cognitivism, and social constructivism. FUN is an easy to remember mnemonic, a practical tool to implement and most importantly, it provides a short checklist to help make teaching and learning fun!



Bibliography
Honey, P. & Mumford, A. (1982) Manual of Learning Styles. London: Peter Honey Publications, London.
Lave, J. & Wenger, E. (1991) Situated Learning: legitimate peripheral participation. Cambridge University Press, New York.
Kaufman, D.M. & Mann, K.V. (2010) Teaching and Learning in Medical Education. In T. Swanwick (Ed) Understanding Medical Education. ASME, Blackwell Publishing.
Knowles MS et al. (1984) Andragogy in Action: applying modern principles of adult learning. Jossey-Bass, San Francisco, CA.
Kolb, D. A. (1984). Experiential learning: Experience as the source of learning and development.
Schön, D. A. (1983). The reflective practitioner: How professionals think in action . New York: Basic Books.
Vygotsky, L. S. (1986). Thought and language (A. Kozulin, Trans.). Cambridge: MIT Press.
Wenger, E. (1998). Communities of practice: Learning, meaning, and identity . Cambridge: Cambridge University Press.

February 16, 2010

Shoulder Exam

Last week I did a presentation at the HOME project on the shoulder exam with a few clinical cases I saw in clinic. It was a smashing success -- everyone seemed to appreciate an introduction/review of some of the maneuvers!

One of the toughest parts of the physical exam is remembering all of the routine shoulder maneuvers.


Here's a few mnemonics I shared that I use to help myself remember things:

Arm Movements

  • Flexion of the arm = Forward to the Face
  • Internal Rotation of the arm = In handcuffs
  • (alternatively, External Rotation = Execution of Ex-Convict)
  • ADduction of the arm = ADDing to the midline
    • Scarf sign = toss a scarf around opposite shoulder

Forearm Movements

  • Supination = Holding a Bowl of "Soup"
  • Pronation = Putting away Palm

Special Maneuvers

  • Range of Motion (and Impingement Signs)
    • Neer = arm "Near" to the ear
    • Hawkins = flap like a "Hawk"
    • Apley scratch test = Applying Suntan lotion to back
  • Rotator Cuff Tears
    • Supraspinatus
      • Jobe's Empty Can (duh!)
    • Hornblower's (ditto!)
  • Biceps
    • Speed's = speedin' down the highway
  • Labral Tear
    • O'Brian = *Irish accent*
      What, so O'Brien gets the thumbs down, but Leno gets a thumbs awp?  I'm cryin' heah!!
  • Subscapularis
    • Gerber's lift off = wiping baby food off the back (okay, its a stretch, but you can't win 'em all!)

 

That's not my second opinion! :)

February 07, 2010

Rheum Case 1

A 55 year old Caucasian male presented to a neighbor island ED with lower extremity skin rash, swelling and severe arthritis following a sore throat.

History of Present Illness:  Pt had an itchy and sore throat for which he was treated with azithromycin.  He sought out new antibiotics after 5 days without relief.  Approximately ten days after the onset of the sore throat, he went to bed complaining of a "sore wrist."  Upon awakening the following morning, he felt severe pain in both wrists with progressive immobility of the left wrist.  His skin broke out with a rash later on in the day on his hands, wrists and ankles.  His legs started swelling and he had difficulty walking.

Denied red eyes or pain with urination.

Past medical history: significant for migratory arthritis, Crohn's disease, HLA-B27(+).  Patient has a long history of severe anaphylactic/eczemal allergic reactions as well.

 

Pertinent physical exam:

Skin exam revealed multiple, progressive, round tender "palpable purpuric" lesions on the hands and ankles.

4+ pitting edema was present in the lower extremities.

 

Diagnosis: enteropathic arthritis and erythema nodosum most likely secondary to a streptococcal infection of the throat with subsequent reaction *HLA-B27(+)

Differential:
Reiter's/Reactive arthritis triad: conjunctivitis, arthritis, urethritis (Can't see, can't pee, can't climb a tree)

Course: Dermatology, ID, and rheumatology consultations were made.  Pt was reluctant to start a course of steroids in the ED, given a past history of steroid-associated insomnia and psychosis.  After 5 days of worsening edema and tender arthritis, he agreed to a steroid burst of 40mg daily and tapering regimen with rheumatology to follow.

 

References

eMedicine: Erythema Nodosum

eMedicine: Enteropathy arthritides


Learning Radiology: An Approach to Arthritis

January 23, 2010

"Nonreassuring" Vocabulary

The word "nonreassuring" is a curious phenomenon of OB/GYN.  Nowhere else is this particular word used in a professional context... which is embarrassing that the word is cited in literature when it doesn't exist.

 

Reassuring is defined thusly:

tr.v. re·as·sured, re·as·sur·ing, re·as·sures

1. To restore confidence to.

2. To assure again.

3. To reinsure.

The implication is that "nonreassuring" means the opposite -- worrisome, vexing.  Of course, to a pregnant mother about to deliver, that's some of the scariest news to hear about her unborn child!

 

In an effort to further obscure the assessment in Fetal Heart Tone monitoring, in April of 2008 the "National Institute of Child Health and Human Development Workshop Report on Electronic Fetal Monitoring" issued a new statement to categorize it in a "Three-Tier Fetal Heart Rate System:

Category I
Category I fetal heart rate (FHR) tracings include all of the following:
• Baseline rate: 110–160 beats per minute (bpm)
• Baseline FHR variability: moderate
• Late or variable decelerations: absent
• Early decelerations: present or absent
• Accelerations: present or absent

Category II
Category II FHR tracings include all FHR tracings not categorized as Category I or Category III. Category II
tracings may represent an appreciable fraction of those encountered in clinical care. Examples of Category II
FHR tracings include any of the following:
Baseline rate
• Bradycardia not accompanied by absent baseline variability
• Tachycardia
Baseline FHR variability
• Minimal baseline variability
• Absent baseline variability not accompanied by recurrent decelerations
• Marked baseline variability
Accelerations
• Absence of induced accelerations after fetal stimulation
Periodic or episodic decelerations
• Recurrent variable decelerations accompanied by minimal or moderate baseline variability
• Prolonged deceleration 2 minutes but 10 minutes
• Recurrent late decelerations with moderate baseline variability
• Variable decelerations with other characteristics, such as slow return to baseline, “overshoots,”
or “shoulders”

Category III
Category III FHR tracings include either:
• Absent baseline FHR variability and any of the following:
- Recurrent late decelerations
- Recurrent variable decelerations
- Bradycardia
• Sinusoidal pattern

Basically, Category I is "reassuring", Category III is "Nonreassuring" (ugh) and Category II is somewhere in between.

 

Personally, I don't like it when the assessment of a patient needs to be translated for the patient's benefit -- why is there a deliberate effort to make something difficult to understand even harder to explain to patients?!?  We don't need to tell someone what they already know, but in a completely different language.

 

It's doubtful, but in the future it could be categorized as "good," "hmm" and "uh oh."

 

DR C BRaVADO: mnemonic for Fetal Heart Monitoring

Define Risk (low/high)
Contractions (freq)
Baseline Rate (110-160)
Variability (10-15bpm)
Accelerations (2, >20 over 20s)
Decelerations (early/variable/late)
Overall assessment (reassuring vs "nonreassuring" ugh!)

 

References:

Advanced Life Support in Obstetrics (ALSO)

AAFP website

AAFP Mnemonics brochure

National Institute of Child Health and Human Development Workshop Report on Electronic Fetal Monitoring

August 22, 2009

PERC up about Well's for PE

PERC up about Well's for PE
mnemonic for workup of Pulmonary Embolism
Items in italics and bold refer to unique features of the PERC score and Well's Criteria.

PERC Score: (PERCx2)
PaO2 <92%
Pulse >100
Extremity swollen now (DVT)
Elderly >50
Recent surgery
Recent DVT -- unilateral extremity swelling in past
Contraceptive use
Coughing up blood


Medscape: Differentiating Low-Risk and No-Risk PE Patients: The PERC Score
http://www.medscape.com/viewarticle/702755

Well's criteria: S.S. PERCC
Suspicious for PE (3)
Signs of DVT now (3)
Pulse >100 (1.5)
Extremity: Past DVT/PE (1.5)
Recent surgery/immob (<4wk/>3d respectively) (1.5)
Coughing up blood (1)
Cancer (1)

High >6.0 pt
Moderate 2.0 to 6.0 pt
Low <2.0 pt


Wells PS, Ginsberg JS, Anderson DR, et al. Use of a clinical model for safe management of patients with suspected pulmonary embolism. Ann Intern Med 1998;129:997-1005. Study used a "minimally invasive" approach to managing patients with suspected PE, emphasizing use of serial dopplers rather than PA grams in patients with a non-diagnostic initial work-up. Approach is comparable to the 1999 ATS guidelines; it does not include CT angiography. A particular strength of the study was the use of set criteria to establish clinical suspicion. http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve&db=pubmed&dopt=Abstract&list_uids=9867786

May 28, 2009

TIMI Score Mnemonic: 1234567

Acute Myocardial Infarction 12 Lead Electrocar...Image via Wikipedia

Thrombolysis in Myocardial/Infarction (TIMI) UA/NSTEMI: 1234567
1/2, 2+, >3+, (4), .05, >65, 7d
  1. CAD hx (>1/2 stenosis)
  2. Angina (2+ episodes in past 24hr)
  3. Atherosclerosis RF (>3+)
    FHx
    CAD
    HTN
    HLD
    DM
    Smoker
  4. Hi cardiac markers (of which there are 4): TnI, TnT, CKMB, LDH
  5. ST-segment deviation of 0.05 mV or more
  6. Age >65
  7. ASA use (in past 7d)

Patients with three or more of the seven variables are considered to be at high risk, whereas those with no more than two of the variables are considered to be at low risk.

 

References:

Optimal Management of Acute Coronary Syndromes. L. D. Hillis and R. A. Lange - 21 May, 2009

TIMI scores :
TIMI Risk Score for UA/NSTEMI
TIMI Risk Score for STEMI

Global Registry of ACS Events (GRACE)
www.outcomes-umassmed.org/grace

Bleeding Risk with www.crusadebleedingscore.org



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May 23, 2009

Chemistries, Part III

This is a follow-up post of the series on Chemistries, Part I and Part II.

I get overwhelmed looking at something like this... so I always take a few deep breaths and approach it sequentially.

Starting with the admission data.
First, I scan it for anything that might KILL the patient. Check the Sodium, Potassium and Glucose levels.

Most alarming is the hyponatremia of 120! Is this something acute or chronic? A sudden drop of sodium may cause seizures and we may be observing a post-ictal state. However, as my astute classmate points out, by circling the blood glucose, we see that the low sodium may in fact be artifactual. When approaching a hyponatremia, we first look to see if the serum osmolality is altered -- Glucose, Alcohol, and Mannitol can cause HYPERtonic hypoNa. For every 100 above 100 of glucose (in this case, 879 or ~900) will draw out water into the intravascular space and dilute the present sodium down by 2.4 (in this case, 9*2.4 = 21.6) So the corrected sodium is actually 120+21.6 or 142. Whew, ok so that was not as scary as I first thought.

Even though we've got a high glucose, I try to stay on task so I don't miss anything. A low potassium. Combined with a low chloride and HCO3, I'd say that this is a volume depleted state with a metabolic acidosis. Normally, we would expect a high potassium with an acidosis as it drives a transcellular shift leaking out potassium. Also, a HYPERglycemic state will also lead to HYPERkalemia. The "paradoxical" hypoK so this is a very significant finding even though it may only be 0.2 below the lower limit of normal. It's not really a paradox, nor is it unexpected. Hang on for further exploration of this.

Let's address the acid-base disorder here.

On the surface, we've got someone with a HCO3 of 18 (which is <24), therefore there is a metabolic acidosis. However, we also note that the anion gap is 32 (which is significantly >>>12!) This is where the "delta-delta" comes into play.

There's all sorts of fancy equations of "it should be greater than this ratio" (d:d>2 = coexisting metabolic acidosis/alkalosis) or "the difference should be less than that" but let's keep things simple, the way my preceptor did.

AG of 32 -> 32-12 = 20. This means that there are 20mEq of unmeasured ions (presumably acid) floating around.
HCO3 of 18 <- for the HCO3 to drop by 20mEq (consumed by the acid in the anion gap, we would need to have an initial HCO3 of 20+18... 38 (>>24!) This markedly high HCO3 is a metabolic alkalosis.

There it is, folks, right there in the numbers: a metabolic alkalosis (likely from vomiting) and a metabolic acidosis (likely from ketone body production.) If you prefer, the d:d is 20:6 or 3.3.



Let's take a deviation in our chemistry analysis for a second....
If we note the time of admission labs as 0930, we can see that the ABG was drawn at 1353. The metabolic acidosis is GONE! It is quite a head-scratcher if this seemingly normal ABG (or only slightly metabolic alkalotic) is the first thing that you see.

Keep in mind though, that the underlying metabolic alkalosis cannot clear as quickly as the metabolic acidosis. We are seeing a cross-sectional sample of a resolving problem:
The tides of acidosis are pulling back, revealing the rocks of alkalosis that were already there, hiding under the surface.

The lesson: BE AWARE OF A WIDE ANION-GAP, even in a non-alkalemic/non-acidemic patient! ESPECIALLY in this seemingly benign situation, because the acid-base see-saw is balanced, but it won't stay there for long!

It's a shame that the ABG was not drawn at the same time as the Chem20.

Now, back to the chemistry analysis...


The differential for an anion-gap metabolic acidosis is classically remembered as MUDPILES: Methanol, Uremia, Diabetic ketoacidosis, Paraldehyde, Isoniazid, Lactic acid, Ethylene glycol, Salicylates. However, Paraldehyde is no longer used. GOLDMARK: Glycols, Oxoproline, L-/D-lactic acid, Methanol, ASA, Renal failure, Ketoacidosis is another mnemonic recommended by Dr. Topf.

Shifting further down the list of labs, we've got an increased creatinine. Sometimes ketones can artificially increase this. It may also reflect a 19:1.31... or ~15:1 prerenal azotemia at work. Decreased renal blood flow from a low volume state like dehydration accounts for this.

Now, the big money. A high glucose and a (+) "large" acetone clinches the diagnosis. As we suspected, from our systematic approach, a patient with hyperglycemia and very little endogenous insulin began to produce other sources of energy for the brain. Namely, proteins like beta-hydroxybutryate and acetoacetate which are acidic. These products are degraded spontaneously into acetone (the same stuff as nail polish remover, giving the patient a nice fruity breath.)

Whew. That's only the initial diagnosis.

We haven't even delved into the treatment and resolution of this common problem.

More to come.

April 20, 2009

Vaccine Schedule Mnemonic

What is a good way to remember the vaccine schedule for children? This can be a pesky task, but I've come up with a pretty succinct way to help you remember!

Don't Be DR. HIP, who flu in an MVA , the DIMVit! and Hit My Teen
@ birth: Be
HepB

@ months 2,4,6: (DR HHIP)
DTaP, Rota, Hib, HepA, IPV, PCV

@ 12 mo-> flu

@ 12mo (MVA)
MMR, VZV, Hep A (w/ booster at least 6 mo later)

@ 4-6 y/o (DIMVit "dimwit")
DTap, IPV, MMR, VZV boosters


Hit My Teen (for teens)
HPV, MCV, Tdap


For adults, just remember a flu vaccine every year, update Td every 10 years (or 5 w/ dirty wounds), PPV @ 65 (or younger with risk factors... PPV, not PCV), and Zostavax (Herpes Zoster vaccine) @ 60 also.

February 17, 2009

Wondering about Wanderers?

Suppose your grandmother wanders out of the house in the middle of the night. What can you do for some peace of mind to keep them safe?

One option is a MedicAlert + Safe Return bracelet. Here's some information:
  • Enroll in program: 1.888.572.8566
  • Report a wandering incident: Call 911 first. Then call 1.800.625.3780. 

  • MedicAlert + Safe Return enrollment package

    For $49.95, with a $25 annual renewal fee, the enrollment kit includes:

    * MedicAlert Identification bracelet or pendant
    * Wallet card
    * "6 Steps to a Safe Return" magnet
    * Personal Health Record Summary
    * Alzheimer's Association brochure

    What are some important considerations for dementia?
    Here's a few mnemonics that might be helpful:
    Remember the reversible causes of DEMENTIA
    Drug intoxication
    EtOH
    Metabolic (hypothyroidism, order TSH!)
    Emotions (screen for depression!)
    Nutrition (Vit B1/B12... order B12!)
    Tumor/Trauma/"Tension"-Normal Pressure Hydrocephalus
    Infection (neurosyphilis, consider RPR!)
    Anemia/Atherosclerosis (order CBC!)


    I'm very fond of important mnemonics that help me quickly recall things at the bedside, especially if they can be easily detected and treated before it progresses into something much more permanent.


    GOMERS go to ground - The Fat Man, House of God. (Elderly people have a tendency to fall.)
    There's a ton of mnemonics on falling. IHATEFALLING, CATASTROPHE*... those are too long but you can google them at your leisure.
    GMA MOVES is more to my liking.
    Gait impairment
    Multiple Meds-Polypharmacy
    Alcohol and drug use
    Medical illness (PNA, MI, anemia, hyponatremia)
    Orthostatic/postprandial hypotension
    Visual Impairment
    Environmental hazards (poor lighting, stairs, rugs, uneven floors)
    Syncope, vertigo, presyncope, or disequilibrium


    References:
    See my post on the Mini Mental Status Exam (ORArL 321 RWD!)
    Medical Mnemonics.com
    Fall Prevention checklists: Patient Handouts

    [edit] * The CATASTROPHE mnemonic is not available on the AAFP article so I've tracked it down for you.
    Functional hx after a fall in the elderly
    Caregiver/housing
    Alcohol (including w/drawal)
    Tx (meds & compliance)
    Affect (depression, lack of initiative)
    Syncope
    Teetering (dizziness)
    Recent illness/hospitalization
    Ocular problems
    Pain w/ mobility
    Hearing
    Environmental hazards

    February 09, 2009

    Gout


    Gout is one of the few arthritides that is reversible and curable with a few simple dietary/lifestyle changes and drugs.

    Current Rheumatology Diagnosis and Treatment elegantly describes it like matches. With flare-ups, the "matches" in the joints light up and cause pain. They can be blown out by NSAIDs, made wet with colchicine and removed entirely with allopurinol. This metaphor is akin to the pathophysiology, with the "matches" representing uric acid crystals precipitating in joint space and lighting up when they get inflamed by the invasion of neutrophils.

    On a more clinical note, here's some mnemonics to remember Gout:

    A TIC ("thick") red, inflamed joint:
    -Trauma, Infection, Crystals

    ROPE in your ddx of arthritis:
    RA, OA, Pseudogout/Psoriatic, Everything else (some crazy zebras: reactive/Reiter's, Still's, Hemochromatosis, Lupus, Lyme, Whipple's, Behcets, Ochronosis)

    When tapping a joint, order 3C's for Synovial Fluid ("CCCnovial")
    Cell Count, Culture, Crystals
    Synovial glucose is not as sensitive as WBC count (>500-1000) for infection.

    The aim for the management of chronic tophaceous gout is <6.0 mg/dL of serum uric acid.
    Think: <6 to fix.

    If the 24 hour urinary uric acid is >800, probenecid can be an alternative to allopurinol. Probenacid is a weak organic acid so it affects the renal levels of other organic acids (ASA, PCN, etc.)
    Think: >800, ProbenAcid instead.

    January 31, 2009

    Study Biases with Eponyms

    Berkson's Bias: selection bias using hospitalized patients as the control.
    Berkson's = Bad/sick patient
    The history: Berkson was a statistician who recognized that small, selective sampling can cause false correlations.
    The problem: "N vs n," statistically.  The data is a poor representative of a greater whole.

    Pygmalion effect: Expectations affect the outcome.
    Pygmalion = "Pointed": hidden agenda, "a self-fulfilling Prophecy"
    The history: Pygmalion is a play in which a professor makes a bet that he can make a poor girl act like a lady. (aka My Fair Lady.)
    The problem: unconsciously, researchers and participants conform to predisposed expectations.

    Hawthorne effect: a tendency for those studied to affect outcome due to their knowledge of the study
    Hawthorne = Heisenberg
    The History: Hawthorne Works was a factory that studied the way lighting affected their workers. Productivity got better when the lights were dimmer. It also got better when the lights were brighter. Soon they realized it was because of the novelty of change (and the presence of researchers.)
    The problem: observing someone affects the way they act.

    January 24, 2009

    Hernias RIP a hole

    What are the borders of the Hasselbach triangle?
    How can you remember the difference between an indirect (50% of all hernias) and a direct (25%) inguinal hernia?

    think: Direct inguinal hernias RIP a hole Directly through the abdominal wall.
    Rectus abdominus
    Inferior epigastric vessels
    Poupart's ligament (also called the inguinal ligament)

    References:
    Surgical Recall

    August 22, 2008

    Trauma Call

    I had trauma call for my Surgery rotation last night. It was traumatic, all right. Not in the gruesome sense of the word, images of human flesh cleaved open by a car, or fecal matter spraying the walls of the OR or anything like that. Let's just get that out of the way.

    It was intellectually traumatic.
    Trauma situations require fast thinking, reflexive actions. They don't spare any moment for deep, ponderous considerations, my usual sort of cognition. I read through a few thick handbooks the night before, nodding off and on around 10-11pm in a meager attempt to prepare myself for the Trauma call.

    We had a Trauma Simulation lab at my medical school in the afternoon, complete with ED physician teachers going through the routine of the Primary Survey: ABCDE -- airway, breathing, circulation, disability, exposure/environment... the stuff we've heard "since grade school." There were some fancy manikins (not mannequins) at the SimTiki lab with different critical conditions resulting from a car crash. Also present: ten medical students with varying degrees of ED experience (ranging from super-star ED ward clerks with trauma experience to ... me.)

    One by one, we were pulled out of the lab to get an assessment with the surgery clerkship coordinator on our TEAM (Trauma Evaluation and Management) skills. I was the first one and we were only five minutes into the first training scenario.

    ... I totally let my simulated patient die in front of me.
    It was tragic. There were some technical difficulties as the manikin needed to be restarted three or four times. In the interim, before the monitors were setup, the surgeon told me -- "continue with your primary survey."

    It went something like this:
    "Uhm... I hear breath sounds, but they sound weird. I don't know how to describe them. Rubbing? Clicking? It's ... not right. Where's my pulse ox and BP data?"
    "The manikin is still being launched. What else are you going to do?"
    "Uhm... I don't know. I can't figure out what those sounds mean. I'm going to continue with Circulation."
    then Disability.
    then Exposure.
    Doh. I clumsily attempted to do some assessments, but I failed to provide any definitive treatment. When the manikin's eyelids closed, the pulse ox data disappeared off the screen and the BP dropped down to the 20s, the overhead voice said in a sardonic tone: "Blood pressure in the 20s is incompatible with life." My failure to diagnose a massive hemothorax with decreased breath sounds, as well as my complete inability to assess the patient properly, led to his eventual demise.

    Luckily for me, it was just a fake patient.
    The shame of the experience, especially with the surgery clerkship coordinator telling me that I needed to read and review everything really hit home. It's one thing to recognize the correct answers; it's another thing entirely to recall it under stress by yourself with a patient changing status before your eyes.

    -----------

    I spent my Trauma call reading through the TEAM handout and dreading the moment when a trauma call came in.

    On the plus side (which is the whole point of the training sim) I learned the Primary Survey by heart! I'll spell out the basics just for fun.

    A: Airway (head and neck)
    -establish airway, put on c-spine
    -foreign objects?
    -if GCS <8, intubate
    B: Breathing and vent (neck and chest)
    -100% O2 for everyone, nasal prongs
    -bag-valve mask if need be or ventilate
    -if lung sounds are reduced, needle thoracostomy!
    C: Circulation and control of hemorrhage (chest and abdomen)
    -BP and EKG
    -hemostasis with direct pressure, inflatable sleeves, etc
    -place two large bore IVs, drop in 2L of LRs for hypotension
    -type and cross for further blood loss
    D: Disability (head and rectum)
    -neuro exam: PERRLA, rectal tone, GCS (E+V+M<=15)
    E: Exposure/Environment
    -Strip clothing
    -Logroll patient, examine back
    -Warm patient (blankets, bair-hugger, fluids, lights)

    SAMPLE history
    S/sx
    Allergies
    Medications
    Past history/Pregnancy
    Last meal
    Events

    Labs/Studies
    ABG
    EKG
    CBC, Chem7
    Type and Cross
    PT/PTT
    UA, UDS
    FAST/DPL
    XR/CT/MRI

    August 11, 2008

    Mini-Mental Status Exam: ORArL 2,3, RWD!

    The components of the Mini-Mental Status Exam (or Folstein test) can be summarized in the quick and easy mnemonic: ORArL 2,3, RWD! It can be a part of a more comprehensive Mental Status Exam performed by psychiatrists as a screening tool assessing cognitive function.

    O: Orientation to Person, Place and Time
    R: Recognition (repeat three objects, i.e. balloon, Pong and happiness)
    A: Attention (Serial 7s counting backward from 100 or spelling WORLD backwards)
    r: recall (ask them to recall the three objects 5 minutes later)
    L: Language
    2: Identify the names of Two objects (pen and glasses/watch)
    3: Follow a Three Step command (take this paper in your right hand, fold it in half and place it on the floor)
    R: Reading (Read this statement and do exactly what it says: "Close your eyes")
    W: Writing (Write a sentence)
    D: Drawing (Copy a figure of two intersecting pentagons)

    This is a useful series to memorize as you become proficient with various components of the MSE since it occupies the majority of your cognitive section. Most people will have a questionaire to fill out, but its easy to do it orally. I find it helpful to create mnemonics of all interview questions since a smooth Q&A session with your patients is a great way to imbue them with confidence in your skills (and any observing residents/attendings.)

    August 10, 2008

    10 parts of the Mental Status Exam (alt: ABC STAMP LICKER)

    The Mental Status Exam is the psychiatric equivalent of a Physical Exam. I struggled to remember all of the components and I found it helpful to break it down into 10 parts for rehearsal. My resident told me about ABC STAMP LICKER after I developed my own method, but I'll share it with you as well.
    1. Appearance
    2. Behavior
    3. Cooperation (note eye contact, degree of friendliness/hostility)
    4. Mood/Affect
    5. Speech
    6. Thought (PCP: Process, Content, Perceptions)
    7. Cognition and Fund
    8. Abstraction (ask "What is meant by 'a rolling stone gathers no moss'?" or "what makes a table and a chair similar?")
    9. Insight
    10. Judgment


    The alternate version (ABC STAMP LICKER):

    • Appearance
    • Behavior
    • Cooperation
    • Speech
    • Thought (Process, Content)
    • Affect
    • Mood
    • Perception (AH/VH)
    • Level of consciousness
    • Insight
    • Cognition
    • Knowledge fund/base
    • Endings (Suicidal, homicidal)
    • Reliability

    Here is a sample student write-up from the UIC Dept of Psychiatry.

    February 21, 2008

    SAFE Moms Take Really Good Care, others go "AAAAA!"

    Antibiotics to avoid in pregnancy:
    SAFE Moms Take Really Good Care
    Sulfonamides
    Aminoglycosides
    Fluoroquinolones
    Erythromycin
    Metronidazole
    Tetracyclines
    Ribavirin
    Griseofulvin
    Chloramphenicol


    Other serious teratogens that make moms go "AAAAA!"
    Alcohol
    Anticonvulsants (Valproic acid, Phenytoin, Carbamazepine)
    Accutane (aka Isoretinoin, Vitamin A)
    Aminopterin
    Androgens


    References
    First Aid for USMLE Step 1
    Wikipedia

    February 07, 2008

    Don't be MEEC about Aseptic Meningitis

    There's no need to be "MEEC" with your differential diagnosis for aseptic meningitis! Here's a mnemonic to help you out.

    Mumps virus
    Echovirus
    Enterovirus (like polio)
    Coxsackie virus

    If an infant comes in with a stiff neck, altered mental sensorium, irritability and high fever, get some CSF. If it's clear and fluid analysis shows elevated protein, but no decrease in glucose and high lymphs... think aseptic meningitis. Bacteria would make it cloudy and eat up the glucose.

    Meningitis is one of those critical ER diagnoses. Don't hesitate or the baby could die! Unless its viral, then there's not much you can do but provide supportive care until it resolves. :-\ Herpesviruses and varicella can be treated with antivirals.

    January 20, 2008

    OLD PAM's menstruation

    Sometimes, young women are reluctant to talk about their female problems. It is important for primary care physicians, then, to ask OLD PAM some screening questions about her menses.

    Onset: When did your menses begin?
    LMP: On what date did you last menstrual periods occur?
    Duration: How often do your menstrual periods occur? How long do they last for?

    Pain: Is there any pain associated with menstruation?
    Activity: Are you sexually active?
    Meds: Are you practicing any form of birth control?

    Questions gathered from
    Seller. Differential Diagnosis of Common Complaints. 4th ed. 2000. (the purple ddx book)

    November 16, 2007

    Saccular aneurysms

    A quick and dirty way to remember the risk factors for developing berry (saccular) aneurysms

    MEN HAS sac(cular aneurysms)
    Marfan's
    Ehler-Danlos
    NF-1
    HTN
    ADPKD
    Smoking

    Berry aneurysms are outpouching of blood vessels that exploit developmental defects hidden in the lining. They occur within the Circle of Willis, a series of connected arteries at the base of your brain that provide collateral blood flow just in case something fails. These puffy red berries can burst and bleed out into the brain causing a hemorrhagic stroke.

    November 15, 2007

    Branches of the ECA & ICA

    Branches of the External Carotid Artery (ECA)
    STAPLe OF PAM ST
    Superior Thyroid
    Ascending Pharyngeal
    Lingual
    Occipital
    Facial
    Posterior Auricular
    Maxillary
    Superficial Temporal

    Branches of the Internal Carotid Artery (ICA)
    OPAAM
    Ophthalmic
    Posterior Communicating
    AChor
    ACA
    MCA


    References
    Moores Clinical Anatomy
    Blumenfeld's Neuroanatomy through Clinical Cases